So Why Not Inform The Public About the Risk Factors For Autism CDC????
The CDC/goverment could have announced in 1958 the warning that older fathers and schizophrenia were connected. They certainly could have done so in 2001. If the pharmaceutical industry/academic research industry is going to profit from an announement the public will hear about it immediately. If the public could prevent schizophrenia, autism, diabetes, Alzheimer's, cancers, mental retardation, in their children through knowing to father babies before 33 the public will not be told and the disorders will be called of "mysterious origin".
Autism and Mystery fits the Interest of Those Who Profit from Autism, Most Psychiatrists in Schizophrenia and Autism research know full well the risk factors.
Remember that childhood schizophrenia is no longer diagnosed since 1994, it is called autism and sporadic schizophrenia is proven to be caused by older men fathering babies. By 33 the sperm stem cells are rapidly accumuating changes in DNA.Women whose father's were older when they were conceived are at a very high risk of having children with genetic disorders. So are people with diabetes, autoimmune thyroid disorders and other autoimmun disease.
Why The Problem with Vaccinations in Autism?
OBJECTIVE: Individuals with schizophrenia and their relatives tend to have either higher or lower than expected prevalences of autoimmune disorders, especially rheumatoid arthritis, celiac disease, autoimmune thyroid diseases, and type 1 diabetes. The purpose of the study was to estimate the association of schizophrenia with these disorders as well as a range of other autoimmune diseases in a single large epidemiologic study. METHOD: The Danish Psychiatric Register, the National Patient Register, and a register with socioeconomic information were linked to form a data file that included all 7,704 persons in Denmark diagnosed with schizophrenia from 1981 to 1998 and their parents along with a sample of matched comparison subjects and their parents. The data linkage required that the autoimmune disease occur before the diagnosis of schizophrenia. RESULTS: A history of any autoimmune disease was associated with a 45% increase in risk for schizophrenia. Nine autoimmune disorders had higher prevalence rates among patients with schizophrenia than among comparison subjects (crude incidence rate ratios ranging from 1.9 to 12.5), and 12 autoimmune diseases had higher prevalence rates among parents of schizophrenia patients than among parents of comparison subjects (adjusted incidence rate ratios ranging from 1.3 to 3.8). Thyrotoxicosis, celiac disease, acquired hemolytic anemia, interstitial cystitis, and Sjögren’s syndrome had higher prevalence rates among patients with schizophrenia than among comparison subjects and also among family members of schizophrenia patients than among family members of comparison subjects. CONCLUSIONS: Schizophrenia is associated with a larger range of autoimmune diseases than heretofore suspected. Future research on comorbidity has the potential to advance understanding of pathogenesis of both psychiatric and autoimmune disorders.
THE GENETICS OF AUTISM
Autism is a severe neurodevelopmental disorder of unknown etiology, with profound consequences for affected individuals and their families. Autism is the classical pervasive developmental disorder (PDD); a group of disorders which also includes Asperger’s syndrome, atypical autism, childhood disintegrative disorder, PDD not otherwise specified (PDDNOS) and Rett syndrome. These disorders are classically defined by a combination of qualitative impairments in three principal areas: verbal and non-verbal communication, reciprocal social interaction, and repetitive and stereotyped patterns of interests and activities.
There is now convincing evidence from twin and family studies for the involvement of genetic factors in the development of autism. The absence of any strong consistent evidence for an environmental, biochemical or neuroanatomical cause has led to an increasing number of genetic studies worldwide to determine the basis of this complex disorder.